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Posted in on June 19, 2010 by
Jake Hanna made a name for himself as a big band drummer with Woody Herman, but when I first ran into him, he had rejoined Marian McPartland for a concert in Manassas, Virginia in 1968. He was then with Merv Griffin, but had worked with Marian over the years and she’d probably begged him to help her out for the Third Battle of Bull Run.
I remember a great deal about that jazz concert, probably because it was the first one I recorded from beginning to end, but there were two things I really remember. One was Bobby Hackett’s onstage diabetic seizure that got everyone’s attention, and the other was a long conversation with Jake about a peculiar incident when he was with Woody Herman.
Jake and I were introduced backstage and over the course of the weekend we talked about this and that. At some point he found out my parent lived in Jamestown, New York. He said he had fond memories of Jamestown, that he’d played a date there with Woody Herman a few years earlier and got into trouble. I told him I thought would be pretty hard, Jamestown isn’t exactly a place known for trouble or even where it could be found without making an effort.
The story was the band played a concert and afterwards some hot little number put the bite on him. Maybe there was a beer or two too many somewhere along the line, but whatever the reason, at some point Jake and the girl found themselves riding in a car, which was pulled over by a local policeman. “What caused him to pull you over,” I asked. Jake’s reply was that he was going the wrong way on a one-way street, and then added, sheepishly, on the sidewalk. Well, maybe it was more than a couple of beers. It got better. It turned out the girl was the daughter of a local FBI agent and was not as old as he thought. I’m sure he told me, but I don’t remember how he talked his way out of that one.
Jake was not only irrepressible, he was an outstanding drummer who was an integral part of our Floating Jazz Festival on a half dozen occasions. In 1990 he was onboard once again, spending most of his time in the North Cape Lounge, with various trios, quartets and vocalists. This was also the year we created special drumheads, based on Paul Bacon’s 1985 Floating Jazz Festival logo design. I had a couple of extras made and asked all the drummers on board the sign them. When he was finished with the magic marker, Jake posed with the drumheads and this is the result.
Jake Hanna aboard the S/S Norway at sea, October 1990
Ford Vox, MD Mon Jan 4, 2010 11:34am EST
The meatus, the opening at the tip of the head where urine and semen are discharged.
Men taking another protease inhibitor ritonavir (Norvir) should not take more than 2.5 mg of vardenafil every 72 hours.
PSA: This test may be abnormal due to prostate enlargement or infection, which may complicate ED.
Penile Injection Therapy Backgrounder - Penile injection therapy was discovered fortuitously. In 1980, the French physician Ronald Virag reported that during penile surgery, he inadvertently injected an anesthetized patient in the wrong part of the penis with papaverine - a substance derived from the opium poppy. The resulting relaxation of the smooth muscle of the penile arterial walls created an unexpected two-hour erection. This mistake prompted serious research into the use of injectable medications to relieve ED. Around the same time, Giles Brindley, a British physiologist and research scientist, discovered that injecting the drug phenoxybenzamine into the corpora cavernosa of the penis could produce an erection within minutes. At a meeting in Paris in 1984, New York urologist Dr. Adrian Zorgniotti presented his first case studies of self-injection utilizing a combination of papaverine and phentolamine. The latter drug blocks the action of neurotransmitters that cause vasoconstriction, causing the smooth muscles of the penis to relax. Two years later, Japanese researchers presented evidence that injecting the drug prostaglandin E-1 produced powerful erections. Slowly, news of the favorable results with the injectable medication began to spread within the small international community of urologists who were treating ED. Most began utilizing all three (papaverine, phentolamine, and prostaglandin E-1) in what was referred to as "trimix."
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Reality: While small amounts of alcohol can lower your inhibitions, heavy drinking can depress your central nervous system and lead to vascular problems, actually increasing your risk for erectile dysfunction.
In DSA, the images are acquired digitally. The computer creates a mask from lower-contrast x-rays of the same area and digitally isolates the blood vessels (this is done manually through darkroom masking with traditional angiography).
The cyclic nucleotide phosphodiesterases (PDEs1) described in this review are a family of related phosphohydrolases that selectively catalyze the hydrolysis of the 3? cyclic phosphate bonds of adenosine and/or guanosine 3?,5? cyclic monophosphate. The structure of cAMP and the bond hydrolyzed is shown in Fig. 1. These enzymes are often referred to as class I cyclic nucleotide PDEs to differentiate them from class II enzymes. Class II enzymes are found in many species including mammals and will also catalyze the hydrolysis of the phosphodiester bond. However, in general, the Class II enzymes do not show the same substrate selectivity as the class I enzymes and much more is known about the class I enzymes.
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Vascular reconstructive surgery Restores natural erections when successful
The physical examination should focus on the vascular, neurological and endocrine systems.28,31 Testes size should be noted and the penis shaft examined to rule out a penile deformity (Peyronie's disease). With respect to laboratory investigations, recommendations vary,30,31 and investigations should follow clinical suspicion of specific disorders. Hemoglobin A1c and serum glucose may be measured to detect occult diabetes, and a lipid screen performed to assess the presence of dyslipidemia. Hormonal screening is controversial; however, it is reasonable to measure testosterone levels in the face of erectile dysfunction combined with loss of libido.28 Patients in whom hyper- or hypothyroidism is suspected should have their thyroid-stimulating hormone level checked.30
Penile implants: Complications include infections in 2%, device malfunction in 4%, erosion of the device through the urethra or skin in 2%, and painful erections in 1%.
Results: Of the 2,010 men interviewed, 257 (12.8%) reported ED. The prevalence increased with age, from 2% in men aged 18-39 to 48% in those >70 years (tested for trend, p = 0.0001). A history of cardiopathy, diabetes, hypertension, neuropathy, thrombotic/hemorrhagic stroke, peripheral vascular disorders, pelvic/medullary injury and pelvic surgery/radiation all increased the risk of ED. The association of hypertension and diabetes tends to increase the risk of ED. In comparison with nondiabetic and nonhypertensive men, the odds ratio (OR) was 1.4 (95% confidence interval (CI), 0.7-3.2) for hypertensive men without diabetes, 4.6 (95% CI, 1.6-13.7) for diabetic men without hypertension and 8.1 (95% CI, 1.2-55.0) for men with diabetes and hypertension. In comparison with never smokers, the OR of ED was 1.7 (95% CI, 1.2-2.4) for current smokers and 1.6 (95% CI, 1.1-2.3) for ex-smokers and increased with duration of the habit.
Metabolism (breakdown) of vardenafil can be slowed by aging, liver disease, and concurrent use of certain medications (such as erythromycin, ketoconazole (Nizoral), and protease inhibitors). Slowed breakdown allows vardenafil to accumulate in the body and potentially increase the risk for side effects. Therefore, in men over age 65, with liver dysfunction, or who are also taking medication(s) that can slow the breakdown of vardenafil, the doctor will initiate vardenafil at low doses to avoid its accumulation. For example:
Common causes of ED include diseases that affect blood flow, such as atherosclerosis (hardening of the arteries); nerve diseases, psychological factors, such as stress, depression, and performance anxiety; and injury to the penis. Chronic illness, certain medications and a condition called Peyronie's disease (scar tissue in the penis) can also cause ED.
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Other adverse events associated with oral PDE-5 inhibitor therapy appear to be few (Table 4). In clinical trials the rates of discontinuation of the drug because of adverse events was low (< 5%) for all of 3 drugs, with few or no cases of priapism.37,53,55,56 Extra caution should be taken for patients who are expected to have reduced clearance of the drug. This group includes men who have severe hepatic or renal insufficiency, men over 65 years old and men taking drugs that inhibit the cytochrome P450 3A4 enzyme (e.g., cimetidine, erythromycin and ketoconazole).57 For these patients it is prudent to start with smaller doses of medication and build up gradually. For sildenafil, it is our experience that a starting dose of 50 mg is safe and effective for all men.
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